Any process that activates or increases the rate, frequency or extent of viral transcription.

Catalysis of movement along a microfilament, coupled to the hydrolysis of a nucleoside triphosphate (usually ATP).

Processes by which a myeloid precursor cell acquires specializes features of an erythrocyte.

The chemical reactions involving beta-amyloid, a glycoprotein associated with Alzheimer's disease, and its precursor, amyloid precursor protein (APP).

Assembly of an actin cortical patch, a discrete actin-containing structure found at the plasma membrane of fungal cells.

Processes by which a myeloid precursor cell acquires specializes features of a megakaryocyte.

The complex that catalyzes the transfer of a glucose moiety from UDP-glucose to a 1,3-beta-D-glucan chain.

Interacting selectively with a SNARE (soluble N-ethylmaleimide-sensitive factor attached protein receptor) protein.

Formation of the actin-based thin filaments of myofibrils.

Formation of the myosin-based thick filaments of myofibrils.

The process by which peroxisomes are delivered to the vacuole and degraded in response to changing nutrient conditions.

The portion of the budding yeast plasma membrane where a daughter cell will emerge. The yeast marks this spot with bud-site selection proteins before bud emergence occurs. Actin is polarized to this spot just prior to and during bud emergence.

establishment of mitotic spindle orientation

mitotic spindle orientation (sensu Saccharomyces)

The processes that set the alignment of mitotic spindle relative to other cellular structures.

Protein complex that plays a role in determining cell polarity by directing the localized assembly of actin filaments at polarization sites; in Saccharomyces the polarisome includes Bni1p, Spa2p, Pea2p, and Bud6p.

Interacting selectively with any carbohydrate.

A cell cycle checkpoint that detects septin defects and responds by inhibiting the mitotic CDK. In Saccharomyces cerevisiae, correct formation of a functional septin cytoskeleton permits the cell to switch to isotropic bud growth and the onset of mitotic chromosome segregation. In the presence of septin defects, the mitotic CDK is inhibited and both the switch to isotropic bud growth and the onset of mitotic chromosome segregation is delayed.