The formation of nucleosomes on newly replicated DNA, coupled to strand elongation.

antigen presentation, endogenous peptide antigen

The process by which antigen-presenting cells express self peptide antigen, via MHC class I molecules, on their cell surface in a form recognizable by lymphocytes.

antigen presentation, exogenous peptide antigen

The process by which antigen-presenting cells express foreign antigen, via MHC class II molecules, on their cell surface in a form recognizable by lymphocytes.

The disassembly of nucleosomes, the beadlike structural units of eukaryotic chromatin composed of histones and DNA.

antigen presentation, endogenous lipid antigen

The process by which antigen-presenting cells express self lipid antigen on their cell surface, via CD1 protein, in a form recognizable by lymphocytes.

Dynamic structural changes to eukaryotic chromatin occurring throughout the cell division cycle. These changes range from the local changes necessary for transcriptional regulation to global changes necessary for chromosome segregation.

antigen presentation, exogenous lipid antigen

The process by which antigen-presenting cells express foreign lipid antigen on their cell surface, via CD1 protein, in a form recognizable by lymphocytes.

platelet-derived growth factor receptor signaling pathway

The series of molecular signals generated as a consequence of a platelet-derived growth factor receptor binding to one of its physiological ligands.

insulin-like growth factor receptor signaling pathway

The series of molecular signals generated as a consequence of the insulin-like growth factor receptor binding to one of its physiological ligands.

Pathways for DNA repair which occur after DNA has replicated, e.g. mismatch repair, and which involve translesion synthesis (TLS-type) DNA polymerases.

The repair of double-strand breaks in DNA via homologous and nonhomologous mechanisms to reform a continuous DNA helix.

double-strand break repair via nonhomologous end-joining

The repair of a double-strand break in DNA in which the two broken ends are rejoined with little or no sequence complementarity. Information at the DNA ends may be lost due to the modification of broken DNA ends.